G6PD POST
Quote from that article about how the G6PD (genetic) Deficiency affects your red blood cells — great basic root cause explanation.
“Pathophysiology
G6PD catalyzes nicotinamide adenine dinucleotide phosphate (NADP) to its reduced form, NADPH, in the pentose phosphate pathway (Figure 14). NADPH protects cells from oxidative damage. Because erythrocytes do not generate NADPH in any other way, they are more susceptible than other cells to destruction from oxidative stress. The level of G6PD activity in affected erythrocytes generally is lower than in other cells. Normal red blood cells that are not under oxidative stress generally exhibit G6PD activity at approximately 2 percent of total capacity. Even with enzyme activity that is substantially reduced, there may be few or no clinical symptoms. A total deficiency of G6PD is incompatible with life.”
“NADPH PROTECTS CELLS FROM OXIDATIVE DAMAGE. CELLS...SUSCEPTIBLE...TO DESTRUCTION FROM OXIDATIVE STRESS.”
This simple fact explains why they have a much shortened life expentency and age aggressively and rapidly. They need help from oxidation.
The question then is would a super anti-oxidant that actually worked help them? What do you think? CURIOUS? (We think the answer is POST )
https://www.aafp.org/afp/2005/1001/p1277.html
