Struggling with stubborn inflammation that just won’t go away? Discover how SOD and Glutathione are essential for detoxing and unlocking true relief—beyond what traditional treatments offer. Ready to get to the root cause of your pain? Visi
Struggling with inflammation or feeling anxious about your genetic testing results? Get expert guidance from Dr. Dan Purser MD and his dedicated Wellness Coaching team. Take control of your health journey—visit PurserWellness.com today!
How Elevated Homocysteine, Impaired Methylation, and Glutathione Deficiency May Be Driving PCOS, Chronic Pain, Brain Fog, Stroke Risk, and Detox Failure — and Why Genetic Testing WILL Change the Way
https://danpursermd.substack.com/p/high-homocysteine-mthfr-mutations?utm_source=youtube
Did you know your body’s natural defense against aging and inflammation quietly declines as you get older? Most people don’t realize that glutathione—the “master antioxidant”—drops with age, genetics, and high homocysteine. The result? More
Superoxide Dismutase 3 (SOD 3), also known as extracellular SOD or ubiquitous SOD, plays a critical role in your body’s defense against inflammation and oxidative stress. Here’s why it matters: Stabilizes Mast Cells SOD 3 helps suppress mast cell activation, reducing the release of inflammatory mediators that contribute to conditions like skin inflammation, inflammatory bowel disease, and chronic pain. Reduces Pro-Inflammatory Cytokines By inhibiting cytokines like TNF-Alpha, SOD 3 can help soothe inflammation at its source. Supports Healthy Joints and Blood Vessels Whether you have joint disc
Homocysteine is bad for you, but it’s a critical part of the transsulfuration pathway. You don’t make glutathione wothout it, but when your levels of homocysteine get too high, it becomes majorly problematic for you.
High homocysteine levels can lead to inflammation, joint and muscle pain, and even shifting pain throughout the body. It’s also linked to birth defects and chronic kidney failure, and can raise blood pressure. Six genetic SNPs (like MTR, MTRR, and CBS) and MTHFR mutations (like C677T) can prevent homocysteine from converting into glutathione, which is essential for keeping inflammation in check. Plus, 12 vitamin deficiencies may temporarily raise homocysteine levels, so addressing these deficiencies is key.
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