The Overlooked Link Between MTHFR Mutations, Hyperhomocysteinemia, PCOS, Fibromyalgia, Chronic Inflammation, and Renal Decline — and Why Precision Genetic Testing May Be the Missing Step
If you have a high homocyine level and you're using glutathione, which is the appropriate thing to do according to the science and all the articles and everything, you got to use a functional glutathione to try and cool off your homoyine level. Let's say it's fixed due to your genetics. You can't bring it down. Nothing will happen. Nothing in
the world can make it come down. So, you're going to always have a high homocyine level. It's genetically related. Check an IL8. Ask your doctor to check a interlucan 8 level to see if it's below three. It means you cooled off your arteries. You cooled off the you prevented the homocyine from causing
the monocytes to release the IL8 and the MCP1. We can assume there's no real way to check an MCP-1 level a huge scientific expense. But IL8 you can easily get I think it's around $50. Check your level, confirm it. You may be on three of the bars capsules or three
of some glutathione or and you might need to take four or five or six or two. [music] That's the way you check it. Iate and look for the value to be less than three [music] and that means that you've act you've actively prevented the homocyine
CCL2 encodes MCP-1, a chemokine that attracts monocytes toward sites of immune activity. Recruitment is a different task from the cytokines that activate those cells once they arrive.
Makes an enzyme that helps produce a form of folate used in the processing of homocysteine. Common variants are different from rare variants that cause severe MTHFR deficiency.
CXCL8 is the gene for IL-8, a chemokine involved in recruiting and activating neutrophils. IL-8 is the protein nickname; CXCL8 is the gene name a DNA report may use.
IL18 encodes interleukin-18, a cytokine that can promote interferon-gamma responses in the appropriate immune context. Its receptor includes components encoded by IL18R1 and IL18RAP.