Let’s explore “IL6”

Still dealing with long COVID symptoms or worried about post-viral inflammation? 🦠 The real damage often comes from a cytokine storm—intense inflammation that raises stroke and heart attack risk. The best defense? Reduced glutathione + SOD.

A subscriber post from Purser Wellness on Substack, covering cytokines and immune signaling, inflammation and antioxidants.

Remember all the talk about cytokine storms a few years back? 🌪️ These intense inflammatory responses are often triggered by overstimulated white blood cells—and low glutathione levels may be the reason why. If you’re dealing with lingering

Struggling with inflammation, high cytokines, or conditions like psoriasis, kidney disease, or chronic fatigue? 🧬 The root cause could be elevated interleukins like IL-8 and MCP-1, often triggered by high homocysteine levels. The most power

MTHFR: THE TERRIBLE - but what is the problem I see with MTHFR SNPs? Inflammation caused by homocysteine! If you have high homocysteine, you get high inflammatory cytokine release. The only way to balance it out is reduced glutathione

High homocysteine levels can lead to inflammation, joint and muscle pain, and even shifting pain throughout the body. It’s also linked to birth defects and chronic kidney failure, and can raise blood pressure. Six genetic SNPs (like MTR, MT

Ever wondered how Homocysteine, MTHFR, and Glutathione are connected? Homocysteine is a highly inflammatory substance that triggers cytokine release in the body, fueling inflammation. It’s part of the methionine cycle, which links directly

A true functional glutathione would help prevent the cytokine cascade that leads to the cytokine storm with the invisible scourge. It would also help ameliorate the inflammatory response to the displaced heme iron we now know is being found

Ask your clinician to define genes/analytes, specimen, assay, repeatability, and how results change management.

Explore the possible links between homocysteine, immune signaling, and pain.

Separate questions about toxin exposure from questions about genetics and antioxidant pathways.

See how IL-8 and MCP-1 feature in research on kidney inflammation and scarring.