What genes coordinate with your intake of folate? The FOLR 1, 2, 3 genes are responsible for the absorption of folate in its different forms. Based on your SNPs for these genes, you may have better affect using different forms of folate. Wh
Homocysteine - a very difficult molecule to keep in balance if you have genetic errors that affect your methylation and transsulfuration pathway. What does that even mean? You aren’t processing glutathione correctly but you can mitigate its
MTHFR: THE TERRIBLE - but what is the problem I see with MTHFR SNPs? Inflammation caused by homocysteine! If you have high homocysteine, you get high inflammatory cytokine release. The only way to balance it out is reduced glutathione and
Can you defeat inflammation at its source? Inflammatory molecules are released in response to a high homocysteine level. That high homocysteine level causes less production of glutathione, causing all kinds of inflammation and problems!
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Homocysteine levels can persistently be elevated if you possess any of seven genetic Single Nucleotide Polymorphisms (SNPs), such as MTHFR, CBS, or FOLH1. When these genes are homozygous, they disrupt the transulfuration pathway, resulting
High homocysteine levels can lead to inflammation, joint and muscle pain, and even shifting pain throughout the body. It’s also linked to birth defects and chronic kidney failure, and can raise blood pressure. Six genetic SNPs (like MTR, MT
Homocysteine, though detrimental to your health, plays a crucial role in the transsulfuration pathway.
While glutathione production is dependent on homocysteine, excessive homocysteine levels pose significant health risks.
Once a fixed le
Why would you have Low Homocysteine? Malnourishment. This is the cause of why you would have a Low Homocysteine - so let’s get a CMA on you and get you feeling GREAT!
Genetics and Homocysteine: What You Should Know
Homocysteine levels can be stubbornly high if you have any of seven genetic SNPs (like MTHFR, CBS, or FOLH1). When these genes are homozygous, they disrupt the transulfuration pathway, leadin
Homocysteine levels can be stubbornly high if you have any of seven genetic SNPs (like MTHFR, CBS, or FOLH1). When these genes are homozygous, they disrupt the transulfuration pathway, leading to high homocysteine, which triggers the releas
Did you know the cause of end-stage renal disease, often left untested, is high homocysteine? When homocysteine levels spike, it triggers the release of damaging cytokines like IL-8 and MCP-1, leading to kidney failure, high blood pressure
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