Let’s explore “Vitamin K1”

Everyone talks about MTHFR. But what if that is only one piece of the puzzle? MTHFR helps process folate, while MTR helps recycle homocysteine back into methionine with the help of vitamin B12. Two genes. Different roles. One interco

A carotenoid that the body can convert to vitamin A.

A fruit used in powders and drinks, often marketed for its vitamin C content.

One member of the vitamin E family; it is distinct from alpha-tocopherol, the form used to establish vitamin E requirements.

Are forms of vitamin K2, a family involved in activation of vitamin K-dependent proteins.

A compound sold in supplements; it is not an established essential human vitamin.

BCO1, also called BCMO1, helps convert beta-carotene from plant foods into retinal, a form used in vitamin A metabolism. Plant carotenoids and preformed vitamin A therefore enter the pathway at different points.

Makes the methionine synthase enzyme, which uses vitamin B12 in the conversion of homocysteine to methionine.

TCN2 helps carry vitamin B12 in the body. A gene result alone cannot tell you your B12 status.

VDR encodes the vitamin D receptor, which helps regulate gene activity in response to active vitamin D. A receptor variant does not by itself mean receptors are absent or that the blood vitamin D level is low.

VKORC1 encodes vitamin K epoxide reductase, part of the vitamin K recycling system used in activating clotting proteins. It is not simply a vitamin K receptor.

ALPL encodes tissue-nonspecific alkaline phosphatase, an enzyme important to bone and tooth mineralization and vitamin B6 metabolism. Persistently low alkaline phosphatase can deserve a different evaluation from a general nutrient deficiency.