Let’s explore “Fasting plasma glucose — lab reference”

Classify by evidence, zygosity, phenotype, and clinical actionability rather than a numeric range.

G6PC2 participates in glucose handling in pancreatic beta cells.

PURSER WELLNESSBenefits of FastingA conversation with Dr. Purser

Dr. Purser shares the benefits of fasting in everyday life. Have you ever tried a 72-hour fast? The benefits of fasting include reduction of premalignant stem cells and chance of cancer. Learn why cancer cells require glucose and how fasting reduces lipids and inflammation. Dr. Purser also discusses the need for careful monitoring of diabetic patients who choose to fast. Learn about precautions of fasting and the need to intake fluids, electrolytes, vitamins, and medications. Contact us at (801) 796-7667 or info@danpursermd.com for help. Check back often for the newest updates!

G6PC2 encodes a glucose-6-phosphatase-related protein expressed in pancreatic islet cells. Common variation is associated with differences in fasting glucose regulation; it is not the insulin receptor.

PURSER WELLNESSMTHFR & The Never-Ending Sugar Crash Cycle: Why You’re Always Tired & Craving SweetsA conversation with Dr. Purser

MTHFR & The Never-Ending Sugar Crash Cycle: Why You’re Always Tired & Craving Sweets If you have an MTHFR variant (C677T or A1298C), you may constantly battle fatigue, sugar cravings, and energy crashes — even when you’re “doing everything right.” In this video, we break down exactly why this happens: How MTHFR/methylation issues impair ATP (energy) production at the cellular level Why your body desperately reaches for quick glucose The vicious cycle of blood sugar spikes → insulin crashes → more cravings & fat storage The downstream effects on antioxidants, DNA repair, and overall energy How to finally break the cycle: Targeted supplementation that fills methylation “holes” (what we learned from 15–20k intracellular tests) Why our MTHFR Daily multivitamin was specifically formulated for this The power of CMA intracellular testing Intermittent fasting (12–16 hrs) Fasted cardio (even just a brisk walk) High-protein, real-food diet with minimal processed sugars If you’re tired of the sugar rollercoaster and want stable energy, this is for you. Links: Read the full Substack article → https://danpursermd.substack.com/p/the-mthfrsugar-craving-cycle Get your CMA intracellular testing & genetics done → Call our office or visit danpursermd.com Shop MTHFR Daily → https://physiciandesigned.com/products/mthfr-daily Have an MTHFR variant? Drop your genotype (heterozygous/homozygous) in the comments — I’m always curious! #MTHFR #SugarCravings #Methylation #ChronicFatigue #Energy #PurserWellness #FunctionalMedicine #IntermittentFasting Follow Dr. Purser! Info: http://www.DanPurserMD.com Supplements: https://www.PhysicianDesigned.com Books: https://www.amazon.com/-/e/B00BIKSGVQ Facebook: https://www.facebook.com/danpursermd Facebook: https://www.facebook.com/PhysicianDesigned Twitter: https://twitter.com/realdanpursermd YouTube: https://www.youtube.com/danpursermd Instagram: https://www.instagram.com/danpurser.md Pinterest: https://www.pinterest.com/danpursermd

Could an overlooked lab marker be providing valuable insight into your health? Understanding your homocysteine level may help you and your healthcare provider identify opportunities to support cardiovascular health, healthy methylation, an

What if one overlooked lab marker is telling you more about your health than you realize? Most people have never heard of homocysteine. Yet when homocysteine levels are elevated, it may signal that critical pathways involving methylation,

👋 Ever stared at your lab results and thought, “What am I even looking at here?” We hear that all the time with our CMA with Redox testing. Patients get TWO pages of results, and without a clear explanation it can feel overwhelming. In th

🚨 High homocysteine isn’t just a lab number—it’s a warning sign. Genes like MTHFR, MTR, MTRR, and GGH can overactivate the methylation cycle and lead to hyperhomocysteinemia, which drives inflammation, stroke risk, and even kidney failure.

Chromium is even more critical than zinc to the glucose transport by the insulin receptor on the cell surface. Intracellular Chromium deficiencies are fairly common in my practice. Again early lack of chromium causes reversible insulin re

Zinc is critical for Glucose Transport through the cell wall via the Insulin Receptor. If you have low intracellular zinc level (much more telling and accurate versus serum levels) then this transport does not occur. Early and milder is j